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Wang ZF; Guo X.
Arsenite-induced apoptosis is prevented by selenite in A375 cell line.
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Biological trace element research
[PubMed: 20390378]
[DOI: 10.1007/s12011-010-8674-5]
Qu L; Gao Y; Sun H; Wang H; Liu X; Sun D.
Role of PTEN-Akt-CREB Signaling Pathway in Nervous System impairment of Rats with Chronic Arsenite Exposure.
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Biological trace element research
[PubMed: 26296331]
[DOI: 10.1007/s12011-015-0478-1]
Chang JS; Yoon IH; Lee JH; Kim KR; An J; Kim KW.
Arsenic detoxification potential of aox genes in arsenite-oxidizing bacteria isolated from natural and constructed wetlands in the Republic of Korea.
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Environmental geochemistry and health
[PubMed: 19548094]
[DOI: 10.1007/s10653-009-9268-z]
Cai L; Liu G; Rensing C; Wang G.
Genes involved in arsenic transformation and resistance associated with different levels of arsenic-contaminated soils.
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BMC microbiology
[PubMed: 19128515]
[DOI: 10.1186/1471-2180-9-4]
Chen S; Shao Z.
Isolation and diversity analysis of arsenite-resistant bacteria in communities enriched from deep-sea sediments of the Southwest Indian Ocean Ridge.
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Extremophiles : life under extreme conditions
[PubMed: 18841325]
[DOI: 10.1007/s00792-008-0195-1]
Ryabinina OP; Subbian E; Iordanov MS.
D-MEKK1, the Drosophila orthologue of mammalian MEKK4/MTK1, and Hemipterous/D-MKK7 mediate the activation of D-JNK by cadmium and arsenite in Schneider cells.
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BMC cell biology
[PubMed: 16451733]
[DOI: 10.1186/1471-2121-7-7]
Effect of Acetyl-L-Carnitine on Antioxidant Status, Lipid Peroxidation, and Oxidative Damage of Arsenic in Rat.
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Biological Trace Element Research
[DOI: 10.1007/s12011-015-0436-y]
Effect of selenomethionine supplementation in food on the excretion and toxicity of arsenic exposure in female mice.
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Biological Trace Element Research
[DOI: 10.1007/s12011-013-9855-9]
Oxidative stress and MAPK involved into ATF2 expression in immortalized human urothelial cells treated by arsenic.
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Archives of Toxicology
[DOI: 10.1007/s00204-013-1058-9]
Arsenic induces apoptosis of human umbilical vein endothelial cells through mitochondrial pathways.
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Cardiovascular Toxicology
[DOI: 10.1007/s12012-010-9073-z]
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